Tag Archives: digestive health

Symptoms of Indigestion

Indigestion centers on recurrent upper-abdominal discomfort. Common features include:

  • Vague pain, burning, or bloating in the epigastrium or left upper quadrant, starting within 1 h after meals or sometimes worsening when the stomach is empty
  • Early satiety: feeling full after only a few bites, preventing completion of a normal meal
  • Post-prandial fullness: bloating persists >1 h after eating and is scarcely relieved by belching or position change
  • Epigastric distension and belching: a subjective sense of abdominal swelling with frequent sour-tasting eructations
  • Nausea or occasional retching; vomitus is usually gastric content without bile or blood
  • Upper epigastric heat: a heartburn-like sensation located higher, easily confused with reflux
  • Loss of appetite, thick tongue coating, halitosis and other ancillary complaints

Symptoms may be intermittent or persist for weeks, often linked to meals, emotion or fatigue and usually remit at night. Warn the doctor if steady weight loss, repeated vomiting, melena or anaemia appears.

Symptom groupTypical descriptionUsual triggers
Epigastric pain/burningDull ache or burning, post-prandial or fastingAcid irritation, mucosal hypersensitivity
Early satietyFullness after small volumeImpaired gastric accommodation
Post-prandial bloatingAbdominal swelling >1 h, belching poorly relievedDelayed emptying, gas pooling
Belching/distensionFrequent eructations, subjective abdominal drumAerophagia, intragastric gas retention
Nausea/retchingOccasional, mostly no bile or bloodDysmotility
Epigastric heatHigher located heartburn-like discomfortAcid or mucosal hypersensitivity
Appetite lossAversion to food or fear of eatingChronic fullness affecting psychology

What Chronic Diarrhea Is Telling You

Chronic diarrhea means >3 loose or watery stools per day for more than four weeks. It is not a disease itself but a common end-point of many mechanisms. Here is a rapid overview from cause to clinic.

1. Osmotic diarrhea

Unabsorbed solutes (lactose, fructose, sorbitol, Mg-antacids) hold water in the lumen; stool output falls markedly when the patient stops eating the offending sugar or salt.

2. Secretory diarrhea

The mucosa actively pours water and electrolytes into the gut; fasting does not stop the flood. Think hyperthyroidism, adrenal insufficiency, gastrinoma, VIPoma, bile-acid malabsorption or chronic alcohol abuse.

3. Malabsorption / steatorrhoea

Insufficient pancreatic enzymes, bile acids or damaged villi (coeliac, short-bowel) leave fat and protein undigested. Stools are bulky, greasy, foul-smelling and often accompanied by weight loss.

4. Inflammatory / exudative diarrhea

Mucosal ulceration leaks protein, blood and pus. Typical offenders: Crohn’s disease, ulcerative colitis, microscopic colitis, radiation enteritis, intestinal tuberculosis, eosinophilic gastro-enteritis.

5. Dysmotility

Rapid transit gives contents too little contact time with the mucosa. Causes include hyperthyroidism, diabetic autonomic neuropathy, post-infectious IBS and pro-kinetic drugs.

6. Dysbiosis & small-intestinal bacterial overgrowth (SIBO)

Broad-spectrum antibiotics, long-term acid suppression or altered anatomy allow colonic-type bacteria to colonise the small bowel, producing gas, altering bile salts and generating watery, bloating diarrhea.

7. Food intolerance & dietary factors

Lactose, fructose, FODMAPs, caffeine, chilli or high-fat meals can trigger osmotic or secretory episodes; true food allergy may add urticaria or eosinophilic infiltrate.

8. Drugs & chronic toxins

NSAIDs, metformin, ACEI/ARB, colchicine, PPIs, Mg-containing antacids, heavy metals, organic phosphorus compounds and chronic alcohol all have “drug-induced or toxic diarrhea” on their label.

9. Systemic disease

Diabetes, hyperthyroidism, adrenal insufficiency, SLE, scleroderma and hypogammaglobulinaemia disturb electrolyte transport or neuromuscular control through endocrine, immune or neural pathways.

10. Neoplasia

Colorectal cancer, lymphoma, VIPoma, medullary thyroid cancer and villous adenoma may secrete peptides, cause partial obstruction or bleed, leading to nocturnal pain, weight loss and anaemia.

Quick look-up table

CategoryClinical tipFirst-line testSimple management
OsmoticStops when patient fastsStool osmotic gapRemove offending sugar / Mg
SecretoryWatery, persists fastingSerum VIP/gastrin, TSHTreat endocrine tumour
SteatorrhoeaGreasy, floats, weight↓Faecal fat, coeliac serologyPancreatic enzymes, gluten-free
InflammatoryBlood/pus, feverColonoscopy + biopsy, faecal calprotectinAnti-inflammatory / immunosuppressant
DysmotilityUrgency, no bloodTSH, HbA1c, transit studyRegulate motility, control glucose
SIBOBloating after mealsBreath test, cultureAntibiotic + probiotic taper
Food intoleranceSymptom food-linkedElimination diaryAvoid trigger diet
Drug-inducedNew drug→new diarrheaDrug history, withdrawalSubstitute or stop drug
SystemicMulti-system signsAuto-screen, endocrine panelTreat primary disease
NeoplasticNocturnal pain, anaemiaTumour markers, imaging, scopeSurgery / chemo / targeted

What Abdominal Pain Is Telling You

Stomach-ache is not just “tummy hurt”. The same spot can mean opposite diseases depending on how the pain feels and what comes with it. Here is a rapid field-map.

1. Map by quadrant

  • Epigastrium: stomach, duodenum, gall-bladder, liver, pancreas
  • Peri-umbilical: small bowel, early appendix, mesenteric nodes
  • Right lower: appendix, right adnexa, right ureter
  • Left lower: sigmoid colon, left adnexa, left ureter
  • Flank / back: kidneys, retro-peritoneum
  • Diffuse: obstruction, peritonitis, metabolic or functional disorders

2. Listen to the “voice” of pain

  • Colicky (waves): spasm of hollow viscus – gastro-enteritis, stones, early obstruction
  • Burning / dull: mucosal lesion – gastritis, reflux
  • Sudden knife-like: perforation – ulcer, diverticulum rupture
  • Persistent boring: capsular stretch – hepatitis, cholecystitis, pancreatitis
  • Referred: appendix umbilicus→RLQ; gall-bladder→right scapula; kidney→groin

3. Decoding companions

  • Nausea / vomiting: infection, pancreatitis, pyloric obstruction
  • Diarrhoea + fever: food-borne infection
  • Jaundice: biliary obstruction or hepatitis
  • Haematuria: urolithiasis
  • Missed period + pelvic pain: ectopic pregnancy, ovarian torsion
  • Zoster rash: neuropathic pain often misdiagnosed

4. Age & sex quick hints

  • Children: intussusception, mesenteric adenitis, Henoch-Schönlein purpura
  • Women of child-bearing age: ectopic pregnancy, corpus luteum rupture, endometriosis
  • Elderly: mesenteric ischaemia, diverticulitis, cancer-related obstruction / perforation

5. Functional vs organic

Pain ≥ 3 months, weekly episodes, normal tests → think irritable bowel or functional dyspepsia; night-pain absent, weight stable, pain relieved by defaecation or flatus.

6. Red-flag call-999 list

Sudden excruciating pain with rigid board-like abdomen, lasting > 6 h, plus high fever, haemodynamic instability, melaena or visible abdominal wall bruising – suspect perforation, massive bleeding, severe pancreatitis or gut necrosis.

At-a-glance table

SiteUsual suspectPain characterKey red-flag add-ons
EpigastriumGastritis / ulcerBurning, meal-relatedAcid regurgitation
Right upperAcute cholecystitisConstant ache → right shoulderFever, positive Murphy sign
Left upperAcute pancreatitisSteady boring to backPersistent vomiting, high serum amylase
Umbilicus → RLQAppendicitisMigrating colic → steadyLocal tenderness, rebound
FlankRenal stoneColic radiates to groinBlood on dip-stick, costo-vertebral angle tenderness
Female pelvisEctopic pregnancySudden stabbingMissed period, shock
GeneralisedBowel obstructionColicky → distensionBile vomiting, absent flatus / stool
VariableIrritable bowelCramp relieved by gasNo weight loss, no night pain

How Did You Catch Gastroenteritis?

a plain-language professional brief

Gastroenteritis doesn’t “fall from the sky.” It happens when the lining of your stomach and intestines is overwhelmed by one or more “attack factors.” Below are the common routes and culprits, followed by a one-page table so you can spot your own risk points at a glance.

1. Microbes: public enemy No. 1

  • Viruses – norovirus, rotavirus, adenovirus; spread faeco-orally or by droplets; famous for cruise-ship, school or nursing-home outbreaks.
  • Bacteria – Salmonella, pathogenic E. coli, Shigella, Campylobacter; linked to under-cooked poultry, eggs, raw seafood or contaminated water.
  • Parasites – Giardia, Cryptosporidium; hide in wild water, swimming pools or pet fur and can cause weeks of diarrhoea.

2. Chemical & drug injury: self-inflicted damage

  • Spirits, strong tea/coffee or super-spicy hot-pot scald the mucosa directly.
  • Regular NSAIDs (aspirin, ibuprofen, diclofenac) weaken the protective barrier and let gastric acid leak in.
  • Broad-spectrum antibiotics wipe out friendly flora, opening the door for C. difficile and pseudomembranous colitis.

3. Eating pattern: the stomach hates “feast-and-famine”

  • One huge meal → acid overshoot and delayed emptying.
  • Skip the next two meals → bile stasis and reflux. Repeat the cycle and mucosal inflammation is guaranteed.

4. Reflux & motility disorders: back-flow from the “drain”

Post-surgical anatomy, gall-bladder disease or chronic constipation can drive bile and pancreatic juice backward into the stomach; if the pylorus is lax, the mix reaches the oesophagus and creates a double acid–alkali burn.

5. Stress & psyche: the brain writes its worry on the gut

Major surgery, trauma, overnight deadlines or chronic anxiety reduce gastric mucosal blood flow and mucus secretion while raising acid output—perfect conditions for erosions and bleeding.

6. Auto-immunity & IBD: friendly fire

Auto-antibodies against parietal cells lead to chronic atrophic gastritis; Crohn’s or ulcerative colitis can ulcerate any part of the GI tract. These “non-infectious” forms tend to relapse for years.

7. Age & radiation: time and the environment gang up

After 60 the stomach makes less acid but also repairs itself more slowly; pelvic or abdominal radiotherapy and some chemo drugs can cause a direct radiation enteritis with diarrhoea and tenesmus.

One-page risk snapshot

Attack typeUsual scene / sourceTypical onsetWho’s most at risk
ViralCruise ships, nurseries, deli saladsAcute watery diarrhoea & vomiting, peak 48 hChildren, elderly, dormitory residents
BacterialRunny eggs, rare beef, tap waterAbdominal pain + fever, bloody mucusRaw-food fans, travellers
ParasiticStreams, pet bowls, poolsDiarrhoea >2 weeks, bloating, gasCampers, toddlers
Drugs / chemicalsPainkillers, antibiotics, spiritsVague pain → erosions → black stoolsChronic users, heavy drinkers
Binge–starve cycleAll-you-can-eat hot-pot plus skipped mealsUpper pain, acid reflux, belchingStudents, busy office staff
Bile refluxPost-GB surgery, chronic constipationBurning, bitter taste, worse after mealsPost-op patients, pregnant women
Psychological stressExams, overtime, sleep debtStomach ache + irregular bowelHigh-pressure jobs, anxious personalities
Auto-immuneNo clear trigger, often with tongue pain & anaemiaChronic atrophy, fatigueMiddle-aged women, family history
Radiation injuryPelvic radiotherapy, chemotherapyTenesmus, mucus-blood stoolsCancer patients